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It is not clear whether reducing TMAO levels can dilute the risk of cardiovascular events in CKD patients (Kim et al., 2016)

Elevated lipid in ovarian tissue and defective lipoprotein processing may generate lipotoxic microenvironments that damage follicle cells and egg viability

When large quantities of NAPQI are formed, hepatocyte glutathione can be critically depleted, meaning that excess NAPQI is not detoxified and cell injury occurs [13]

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