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counterintuitively, this step is slowed down by mechanisms outlined below, in order to generate an upstream metabolic traffic jam and help divert glycolytic intermediates to side pathways yielding NADPH, hexosamines, serine, glycine, TGs, PLs, etc

May 21 2024;16(11)doi:10.3390/nu16111550 McNulty H, Pentieva K, Ward M

221,222 Activation of Akt stimulates -cell proliferation, reduces apoptosis, and enhances insulin secretion by regulating downstream effector molecules like Forkhead box protein O1 (FoxO1) and the glucose transporter type 2 (GLUT2)

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